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Conceptually, reperfusion after decompression in rats that had intra-abdominal hypertension and occlusion/occlusion-like syndrome resolved the implementation [8,9,10,11,12,13,14,15,16,17,18,19,20,21,22,23,24,25] of Robert’s long-standing concept of cytoprotection positing that the cells afforded by a cytoprotection agent should equally resist noxious events [35], whether ischemia or reperfusion, and thereby stands BPC 157 therapy as a particular cytoprotective agent’s effect [1,2,3,30,31,32,33,34]