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In consequence, with the thrombocytopenia and haemorrhage, the expected lack of NO-suppression of platelet aggregation [33], the acceleration and amplification of the induced pattern of platelet activation resulting in platelet exhaustion [9] (L-NAME), grossly correspond to the suggested inhibition of platelet aggregation, by direct action on an intraplatelet constitutive calcium-dependent NOS [33–35] (L-arginine)

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10.1254/jphs.fp0061322 [DOI] [PubMed] [Google Scholar] Drmic D., Samara M., Vidovic T., Malekinusic D., Antunovic M., Vrdoljak B., et al

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